Archive for the ‘Case Studies’ Category

Weight Loss Products

Thursday, September 22, 2011 @ 07:09 PM
posted by

Are all weight loss products the same?

There are hundreds of weight loss products on the market. With all of the hype, it’s impossible to tell what’s what. I decided to research this in order to compile a list of the most popular products and their effectiveness, in an effort to cut through all the hype and false information out there. The surprising thing I found is that once you look more closely, there are really only a handful of substances out there–including herbs, amino acids, enzymes, etc–that make up the ingredients in the vast majority of over-the-counter (OTC) weight loss products. They are just being sold under a lot of different names, with a big variety in price and marketing. So here is the list of products, which I hope you will find helpful.

Stimulants - by far the most popular category

Ephedra (Ma Huang)

This stimulant does work well for some things. Chinese herbalists use it to treat respiratory infections, and ephedrine (a derivative) is in most asthma medications and allergy pills because it has powerful bronchodilation properties. But as a weight loss aid this is not the way to go, even though this stimulant is probably the most common ingredient in all weight loss products. It is often combined with caffeine and aspirin, known as an E/C/A stack. This is basically like taking speed. (There is only a slight difference chemically between methamphetamine and ephedrine.) The idea is it suppresses appetite, thus leading to weight loss. But there are many side effects, such as irregular or accelerated heart rate, insomnia, elevated blood pressure, shortness of breath, seizures, etc. It is extremely misleading to label products with these stimulants “all natural” because then people think they must be harmless. Too much of this stuff can be very dangerous, even fatal, especially if you have high blood pressure or heart problems. (Ephedra is natural in the same sense that arsenic is also natural.) Even if stimulants like Ephedra do work for you temporarily, research has shown that you gain 100% of the weight back once you stop taking it.

Other Herbal Stimulants

 The bottom line is that although stimulants can aid in minimizing caloric intake, the main effect is water loss, not real fat loss. Plus they are very addicting, yet extremely unhealthy to take long-term. Stimulants are not an effective way to boost metabolism either, no matter what the label says; exercise is much better at achieving this.

Guarana ****

Made from the seeds of a plant native to Brazil, this is a strong stimulant that has a high amount of caffeine. Side effects from guarana may include nausea, dizziness, insomnia, anxiousness, etc.

Yerba Maté (also known as Paraguay tea)

A strong central nervous system stimulant that acts very much like caffeine. It has not been proven to aid in weight loss but can cause high blood pressure, insomnia, and other side effects associated with stimulants.

Kola Nut

The seed kernel of a large African tree, this is the active ingredient in Coca Cola and contains caffeine.

Bitter Orange (Synephrine)

The active ingredient is called synephrine, which is similar to ephedrine.

Thermogenics

These products often contain stimulants such as ephedrine or caffeine (see description on previous page), but may also include other ingredients that can decrease fat absorption and/or increase muscle mass. (Some of these ingredients will be covered separately later in this article.) The claim is that thermogenics increase your metabolism, but the only proven and safe way to do this is through exercise.

Leptoprin (Anorex)

This product is made up primarily of the E/C/A stack of ephedra, caffeine and aspirin, and is not really different than many other so called thermagenic products on the market. However, this product sells like crazy, so I wanted to investigate. It’s main claim to fame is that it’s very expensive ($153/bottle). In fact, that seems to be the focus of the ad campaign for this product: it’s so costly that it must be good. Their TV commercials say things like’”it’s much too powerful for the casual dieter.” Refer to the section on stimulants as to why to stay away from products like this.

More “Miracle” Products

Chitosan
This is a derivative of chitin, which is a substance found in the shells of sea creatures such as shrimp, lobsters, and crabs. It cannot be digested and passes through your body without adding any calories. It is supposed to reduce fat absorption because its chemical nature makes it to bind with fatty foods, removing some of the fat from your body. However, studies have found it no more effective in weight loss than a placebo. And according to a study by HealthWatch, an independent watch-dog group, there was no measurable increase in the amount of fat excreted after taking this product. In addition, it may be dangerous, by inhibiting your body’s ability to absorb certain nutrients such as the fat-soluble vitamins A and D and certain phyto-chemicals found in vegetables and fruit.

CLA – Conjugated Linoleic Acid
This fatty acid is supposed to speed up fat metabolism and increase muscle mass. Not yet proven for weight loss, CLA can cause flatulence and gastrointestinal discomfort.

Chromium (GTF Chromium/Chromium Picolinate)  ****
This is a trace mineral that may help reduce sugar cravings and regulate appetite by stabilizing blood sugar levels. The evidence on chromium as a weight loss aid is still inconclusive, but some health practitioners do advocate it as part of an entire weight loss program. It won’t help you lose weight by itself, but if you take it along with changing your diet and adding exercise, it may be helpful. (Make sure to follow the recommended dosage, as an overdose of this mineral can lead to kidney damage.)

GLA – Gamma-Linolenic Acid
Found in evening primrose oil, GLA is an essential fatty acid advocated by some, such as nutritionist Ann Louise Gittleman, as helping to burn off extra calories and boost energy.

HCA – Hydroxycitric Acid ****
This is the active ingredient in Garcinia Cambogia, which is an evergreen tree from Southeast Asia. According to Dr. Andrew Weil, some data from animal studies suggest HCA may suppress appetite and the formation of fats and cholesterol in the liver.

5-HTP (5-Hydroxytryptophan)
This is supposed to help you lose weight by raising the levels of serotonin, which can influence eating behavior. The body turns the amino acid tryptophan into 5-HTP. (Tryptophan was banned by the FDA due to health risks.) 5-HTP can have bad interactions with certain drugs, and is harmful to women who are pregnant or nursing. It is not a good idea to take this substance without consulting your doctor first.

L-Carnitine
an amino acid claimed to help with weight loss by increasing energy and lean muscle mass. So far the jury is still out on this one.

Pyruvate (Pyruvic Acid)
This is a naturally occurring enzyme that forms in the body during digestion of carbohydrates and protein. Some products containing this ingredient claim that it is a natural alternative to Fen-phen (fenfluramine), but a report in the International Journal of Sports Nutrition claims such statements false and misleading. (Fen-phen was extremely popular, but banned by the FDA in 1997 due to heart problems developing in some people who took the product.) There has been at least one study where researchers found pyruvate no more effective than a placebo, and side effects can include intestinal distress, bloating, and diarrhea. According to Dr. Andrew Weil, “pyruvate does not cross easily from our blood into our cells, so even taking large amounts of it will have little or no effect in burning off of fat. At best, it is just another diet pill ‘miracle’ that will only slim down your wallet.”

PPA (Phenylpropanolamine)
Never proven effective in weight loss, PPA was banned in 2000 by the FDA from being sold without a prescription because it was deemed too dangerous. At the time it was the active ingredient in Dexatrim, Acutrim and several other OTC products. It can cause headaches, extreme spikes in blood pressure, stroke, and even death in some cases.

Herbal Diuretics
Very common in OTC weight loss products, diuretics can trick you into thinking you’re getting thinner, when all you are really doing is losing water weight. Herbal diuretics include hawthorn, dandelion, juniper seeds, horse tail, shave grass, and green tea. Most of these herbs are not toxic, but some can have bad interactions with prescription medications. And if you abuse these products, too much water loss will deplete your body of sodium and potassium. A much healthier way to achieve the same effect is to drink lots of purified water. You will lose excess water weight, hydrate your skin, and get other health benefits as well.

Other Herbs for Weight Loss

Bladderwrack 

This is a kind of seaweed that contains a high amount of iodine, which may stimulate the thyroid. Unless you know you have an iodine deficiency, it is not a really good idea to take this.

St. John’s Wort\

Primarily used to fight depression, this herb has not been proven effective for weight loss and can actually be dangerous. If you take St. John’s wort, you need to avoid certain medications such as Prozac, Zoloft and Paxil, and foods containing tyramine such as wine, cheese and aged meats.

White Willow Bark

This herb contains the active ingredient in aspirin. It may work for a headache, but it is not proven effective for weight loss.

Dietary Fiber
The idea behind taking products with dietary fiber is that they will make you feel full, so you supposedly eat less. They also aid in elimination, which can help with detoxification.

Pectin

This is a soluble fiber that occurs naturally in such fruits as apples, pears, and bananas. Pectin’s actual effect on appetite may vary for different people.

Guar Gum

A dietary fiber obtained from the Indian cluster bean, guar gum is used in many foods as a thickening agent. This fiber is supposed to make you feel full like pectin, but it can cause gastrointestinal disorders and fluctuations in blood sugar levels.

Glucomannan 

Derived from the konjac root, it can absorb a great deal of water and thus promote a feeling of fullness.

Fenugreek Seeds 

Contain 40% soluble fiber, which has the same effects as listed above. Fenugreek seeds may also reduce blood glucose levels.

Does fat mean unfit?

Wednesday, August 17, 2011 @ 02:08 PM
posted by

 

fitResearchers have found that fat (obese) men (who have been diagnosed withmetabolic syndromecan keep their arteries healthy and preventcardiovascular disease, heart attack and stroke by maximizing theircardiovascular fitness.

The Fitness Study

Scientists looked at a group of men with varying levels of cardiovascularfitness and metabolic syndrome.

  • Cardio fitness was measured by peak oxygen uptake during a standard treadmill test
  • Risk for heart disease was measured by their level of arterial stiffness.

Not too surprisingly, the researchers found that the men diagnosed with metabolic syndrome had ,on average, higher levels of arterial stiffness.

However, when the men were separated according to their levels of cardiovascular fitness, they found that all of the men in the highest quartile of fitness had significantly lower levels of arterial stiffness than those men in the lowest quartile of fitness. That included fit guys who had been diagnosed with metabolic syndrome.

In fact, the fit and fat guys had the same levels of arterial stiffness as their skinny yet exercise-adverse brothers.

Conclusion

It is possible for fat guys to be fit guys.

.

Unfortunately, it’s unlikely that those fat & fit guys will ever be as fit as their lean & fit workout buddies.

.

 

See more information on how you can stay fit at http://myweightcare.com/category/fitness-and-workout-programs/

Sugar is killing us all!

Monday, August 8, 2011 @ 02:08 PM
posted by
Death by sugar may not be an overstatement—evidence is mounting that sugar is THE MAJOR FACTOR causing obesity and chronic disease. We need to get back the healthy eating and this post will show you why.

Dr. Mercola’s Comments:
Follow Dr. Mercola on Twitter Follow Dr. Mercola on Facebook

Is sugar a sweet old friend that is secretly plotting your demise?

There is a vast sea of research suggesting that it is. Science has now shown us, beyond any shadow of a doubt, that sugar in your food, in all its myriad of forms, is taking a devastating toll on your health.

The single largest source of calories for Americans comes from sugar—specifically high fructose corn syrup. Just take a look at the sugar consumption trends of the past 300 years:[1]

  • In 1700, the average person consumed about 4 pounds of sugar per year.
  • In 1800, the average person consumed about 18 pounds of sugar per year.
  • In 1900, individual consumption had risen to 90 pounds of sugar per year.
  • In 2009, more than 50 percent of all Americans consume one-half pound of sugar PER DAY—translating to a whopping 180 pounds of sugar per year!

Sugar is loaded into your soft drinks, fruit juices, sports drinks, and hidden in almost all processed foods—from bologna to pretzels to Worcestershire sauce to cheese spread. And now most infant formula has the sugar equivalent of one can of Coca-Cola, so babies are being metabolically poisoned from day one if taking formula.

No wonder there is an obesity epidemic in this country.

Today, 32 percent of Americans are obese and an additional one-third are overweight. Compare that to 1890, when a survey of white males in their fifties revealed an obesity rate of just 3.4 percent. In 1975, the obesity rate in America had reached 15 percent, and since then it has doubled.

Carrying excess weight increases your risk for deadly conditions such as heart disease, kidney disease and diabetes.

In 1893, there were fewer than three cases of diabetes per 100,000 people in the United States. Today, diabetes strikes almost 8,000 out of every 100,000 people.[1]

You don’t have to be a physician or a scientist to notice America’s expanding waistline. All you have to do is stroll through a shopping mall or a schoolyard, or perhaps glance in the mirror.

Sugars 101 — Basics of How to Avoid Confusion on this Important Topic

 

It is easy to become confused by the various sugars and sweeteners. So here is a basic overview:

  • Dextrose, fructose and glucose are all monosaccharides, known as simple sugars. The primary difference between them is how your body metabolizes them. Glucose and dextrose are essentially the same sugar. However, food manufacturers usually use the term “dextrose” in their ingredient list.
  • The simple sugars can combine to form more complex sugars, like the disaccharide sucrose (table sugar), which is half glucose and half fructose.
  • High fructose corn syrup (HFCS) is 55 percent fructose and 45 percent glucose.
  • Ethanol (drinking alcohol) is not a sugar, although beer and wine contain residual sugars and starches, in addition to alcohol.
  • Sugar alcohols like xylitol, glycerol, sorbitol, maltitol, mannitol, and erythritol are neither sugars nor alcohols but are becoming increasingly popular as sweeteners. They are incompletely absorbed from your small intestine, for the most part, so they provide fewer calories than sugar but often cause problems with bloating, diarrhea and flatulence.
  • Sucralose (Splenda) is NOT a sugar, despite its sugar-like name and deceptive marketing slogan, “made from sugar.” It’s a chlorinated artificial sweetener in line with aspartame and saccharin, with detrimental health effects to match.
  • Agave syrup, falsely advertised as “natural,” is typically HIGHLY processed and is usually 80 percent fructose. The end product does not even remotely resemble the original agave plant.
  • Honey is about 53 percent fructose[2], but is completely natural in its raw form and has many health benefits when used in moderation, including as many antioxidants as spinach.
  • Stevia is a highly sweet herb derived from the leaf of the South American stevia plant, which is completely safe (in its natural form). Lo han (or luohanguo) is another natural sweetener, but derived from a fruit.

All Sugars are Not Equal

Glucose is the form of energy you were designed to run on. Every cell in your body, every bacterium—and in fact, every living thing on the Earth—uses glucose for energy.

But as a country, sucrose is no longer the sugar of choice. It’s now fructose.

If your diet was like that of people a century ago, you’d consume about 15 grams per day—a far cry from the 73 grams per day the typical person gets from sweetened drinks. In vegetables and fruits, it’s mixed in with vitamins, minerals, enzymes, and beneficial phytonutrients, all which moderate the negative metabolic effects. Amazingly, 25 percent of people actually consume more than 130 grams of fructose per day.

Making matters worse, all of the fiber has been removed from processed foods, so there is essentially no nutritive value at all. And the very products most people rely on to lose weight—the low-fat diet foods—are often the ones highest in fructose.

It isn’t that fructose itself is bad—it is the MASSIVE DOSES you’re exposed to that make it dangerous.

There are two overall reasons fructose is so damaging:

  1. Your body metabolizes fructose in a much different way than glucose. The entire burden of metabolizing fructose falls on your liver.
  2. People are consuming fructose in enormous quantities, which has made the negative effects much more profound.

The explosion of soda consumption is the major cause of this. Healthy eating requires we stop this epidemic and get back to basics and get away from simple sugars.

Today, 55 percent of sweeteners used in food and beverage manufacturing are made from corn, and the number one source of calories in America is soda, in the form of high fructose corn syrup.

Food and beverage manufacturers began switching their sweeteners from sucrose to corn syrup in the 1970s when they discovered that HFCS was not only far cheaper to make, it’s about 20 percent sweeter than conventional table sugar that has sucrose.

HFCS contains the same two sugars as sucrose but is more metabolically risky to you, due to its chemical form.

The fructose and the glucose are not bound together in HFCS, as they are in table sugar, so your body doesn’t have to break it down. Therefore, the fructose is absorbed immediately, going straight to your liver.

Too Much Fructose Creates a Metabolic Disaster in Your Body

Dr. Robert Lustig, Professor of Pediatrics in the Division of Endocrinology at the University of California, San Francisco, has been a pioneer in decoding sugar metabolism. His work has highlighted some major differences in how different sugars are broken down and used by the human body.

I highly recommend watching Lustig’s lecture in its entirety if you want to learn how fructose is ruining your health biochemically.

As I mentioned earlier, after eating fructose, most of the metabolic burden rests on your liver. This is NOT the case with glucose, of which your liver breaks down only 20 percent. Nearly every cell in your body utilizes glucose, so it’s normally “burned up” immediately after consumption.

So where does all of this fructose go, once you consume it?

Onto your thighs. It is turned into FAT (VLDL and triglycerides), which means more fat deposits throughout your body.

Eating Fructose is Far Worse than Eating Fat

However, the physiological problems of fructose metabolism extend well beyond a couple of pant sizes:

  • Fructose elevates uric acid, which decreases nitric oxide, raises angiotensin, and causes your smooth muscle cells to contract, thereby raising your blood pressure and potentially damaging your kidneys.[1]Increased uric acid also leads to chronic, low-level inflammation, which has far-reaching consequences for your health. For example, chronically inflamed blood vessels lead to heart attacks and strokes; also, a good deal of evidence exists that some cancers are caused by chronic inflammation. (See the next section for more about uric acid.)
  • Fructose tricks your body into gaining weight by fooling your metabolism—it turns off your body’s appetite-control system. Fructose does not appropriately stimulate insulin, which in turn does not suppress ghrelin (the “hunger hormone”) and doesn’t stimulate leptin (the “satiety hormone”), which together result in your eating more and developing insulin resistance.[3] [4]
  • Fructose rapidly leads to weight gain and abdominal obesity (“beer belly”), decreased HDL, increased LDL, elevated triglycerides, elevated blood sugar, and high blood pressure—i.e., classic metabolic syndrome.
  • Fructose metabolism is very similar to ethanol metabolism, which has a multitude of toxic effects, including NAFLD (non-alcoholic fatty liver disease). It’s alcohol without the buzz.

These changes are not seen when humans or animals eat starch (or glucose), suggesting that fructose is a “bad carbohydrate” when consumed in excess of 25 grams per day. It is probably the one factor responsible for the partial success of many “low-carb” diets.

One of the more recent findings that surprised researchers is that glucose actually accelerates fructose absorption, making the potential health risks from HFCS even more profound.[1]

You can now see why fructose is the number one contributing factor to the current obesity epidemic.  To stop the obesity epidemic start eating healthy natural foods.

Is Uric Acid the New Cholesterol?

By now you are probably aware of the childhood obesity epidemic in America—but did you know about childhood hypertension?

Until recently, children were rarely diagnosed with high blood pressure, and when they were, it was usually due to a tumor or a vascular kidney disease.

In 2004, a study showed hypertension among children is four times higher than predicted: 4.5 percent of American children have high blood pressure. Among overweight children, the rate is 10 percent. It is thought that obesity is to blame for about 50 percent of hypertension cases in adolescents today.[1]

Even more startling is that 90 percent of adolescents who have high blood pressure have elevated uric acid levels.

This has led researchers to ask, what does uric acid have to do with obesity and high blood pressure?

In his book, The Sugar Fix: The High-Fructose Fallout That is Making You Fat and Sick, DrRobert J. Johnson makes a compelling argument for a previously unrecognized connection between excess sugar consumption and high uric acid levels. However, he promotes artificial sweeteners as an alternative to sugar and makes other recommendations that I don’t agree with. Healthy Eating of low sugar foods with the correct natural sweeteners or none at all are a better choice.

Dr. Johnson is a conventional physician who has not accepted large parts of natural medicine, however, he is one of the leading researchers defining the extent of fructose toxicity.  He has spent many years of his life dedicating himself to uncover this mystery.

There are more than 3,500 articles to date showing a strong relationship between uric acid and obesity, heart disease, hypertension, stroke, kidney disease, and other conditions. In fact, a number of studies have confirmed that people with elevated serum uric acid are at risk for high blood pressure, even if they otherwise appear to be perfectly healthy.

Uric acid levels among Americans have risen significantly since the early half of the 20th Century. In the 1920s, average uric acid levels were about 3.5 ml/dl. By 1980, average uric acid levels had climbed into the range of 6.0 to 6.5 ml/dl and are probably much higher now.

How Does Your Body Produce Uric Acid?

It’s a byproduct of cellular breakdown. As cells die off, DNA and RNA degrade into chemicals called purines. Purines are further broken down into uric acid.

Fructose increases uric acid through a complex process that causes cells to burn up their ATP rapidly, leading to “cell shock” and increased cell death. After eating excessive amounts of fructose, cells become starved of energy and enter a state of shock, just as if they have lost their blood supply. Massive cellular die-off leads to increased uric acid levels.

And cells that are depleted of energy become inflamed and more susceptible to damage from oxidative stress. Fat cells actually become “sickly,” bloating up with excessive amounts of fat.

There is a simple, inexpensive blood test for determining your uric acid level, which I recommend you have done as part of your routine health checkups. Your level should be between 3.0 and 5.5 mg/dl, optimally.

There is little doubt in my mind that your uric acid level is a more potent predictor of cardiovascular and overall health than your total cholesterol level is. Yet virtually no one is screening for this.

Now that you know the truth you don’t have to be left out in the cold, as this is a simple and relatively inexpensive test that you can get at any doctor’s office. Odds are very good your doctor is clueless about the significance of elevated uric acid levels, so it will not likely be productive to engage in a discussion with him unless he is truly an open-minded truth seeker.

Merely get your uric acid level, and if it is over 5 then eliminate as much fructose as you can (also eliminate all beer), and retest your level in a few weeks.

Sugar Sensitization Makes the Problem Even WORSE!

There is yet another problem with sugar—a self-perpetuating one.

According to Dr. Johnson1, sugar activates its own pathways in your body—those metabolic pathways become “upregulated.” In other words, the more sugar you eat, the more effective your body is in absorbing it; and the more you absorb, the more damage you’ll do.

You become “sensitized” to sugar as time goes by, and more sensitive to its toxic effects as well.

The flip side is, when people are given even a brief sugar holiday, sugar sensitization rapidly decreases and those metabolic pathways become “downregulated.” Research tells us that even two weeks without consuming sugar will cause your body to be less reactive to it.

Try it for yourself! Take a two-week sugar sabbatical and see how different you feel.

Are Fruits Good or Bad for You?

Keep in mind that fruits also contain fructose, although an ameliorating factor is that whole fruits also contain vitamins and other antioxidants that reduce the hazardous effects of fructose.

Juices, on the other hand, are nearly as detrimental as soda, because a glass of juice is loaded with fructose, and a lot of the antioxidants are lost.

It is important to remember that fructose alone isn’t evil as fruits are certainly beneficial. But when you consume high levels of fructose it will absolutely devastate your biochemistry and physiology. Remember the AVERAGE fructose dose is 70 grams per day which exceeds the recommend limit by 300 percent.

So please BE CAREFUL with your fruit consumption. You simply MUST understand that because HFCS is so darn cheap, it is added to virtually every processed food. Even if you consumed no soda or fruit, it is very easy to exceed 25 grams of hidden fructose in your diet.

If you are a raw food advocate, have a pristine diet, and exercise very well, then you could be the exception that could exceed this limit and stay healthy.

Dr. Johnson has a handy chart, included below, which you can use to estimate how much fructose you’re getting in your diet. Remember, you are also likely getting additional fructose if you consume any packaged foods at all, since it is hidden in nearly all of them.

Fruit Serving Size Grams of Fructose
Limes 1 medium 0
Lemons 1 medium 0.6
Cranberries 1 cup 0.7
Passion fruit 1 medium 0.9
Prune 1 medium 1.2
Apricot 1 medium 1.3
Guava 2 medium 2.2
Date (Deglet Noor style) 1 medium 2.6
Cantaloupe 1/8 of med. melon 2.8
Raspberries 1 cup 3.0
Clementine 1 medium 3.4
Kiwifruit 1 medium 3.4
Blackberries 1 cup 3.5
Star fruit 1 medium 3.6
Cherries, sweet 10 3.8
Strawberries 1 cup 3.8
Cherries, sour 1 cup 4.0
Pineapple 1 slice
(3.5″ x .75″)
4.0
Grapefruit, pink or red 1/2 medium 4.3
Fruit Serving Size Grams of Fructose
Boysenberries 1 cup 4.6
Tangerine/mandarin orange 1 medium 4.8
Nectarine 1 medium 5.4
Peach 1 medium 5.9
Orange (navel) 1 medium 6.1
Papaya 1/2 medium 6.3
Honeydew 1/8 of med. melon 6.7
Banana 1 medium 7.1
Blueberries 1 cup 7.4
Date (Medjool) 1 medium 7.7
Apple (composite) 1 medium 9.5
Persimmon 1 medium 10.6
Watermelon 1/16 med. melon 11.3
Pear 1 medium 11.8
Raisins 1/4 cup 12.3
Grapes, seedless (green or red) 1 cup 12.4
Mango 1/2 medium 16.2
Apricots, dried 1 cup 16.4
Figs, dried 1 cup 23.0

In addition to limiting your intake of fructose, you should eliminate all sweetened beverages and fruit juices (including all artificial sweeteners) and drink only pure water and raw milk.

You can buy pure glucose (dextrose) as a sweetener for about $1 a pound. It is only 70% as sweet as sucrose, so you’ll end up using a bit more of it for the same amount of sweetness, making it slightly more expensive than sucrose—but still well worth it for your health as it has ZERO grams of fructose.

Remember that glucose can be used directly by every cell in your body and as such is far safer than the metabolic poison fructose.

Beer is also a good beverage to AVOID since it increases uric acid levels, just like fructose does, resulting in many of the same toxic effects.

All alcoholic beverages cause you to produce excess uric acid (and block your kidneys from excreting it), but beer seems to have a more pronounced effect on uric acid levels because it’s a rich source of guanosine, the type of purine that is most readily absorbed by the body.1

76 Additional Ways Sugar Can Ruin Your Health

In addition to throwing off your body’s homeostasis and wreaking havoc on your metabolic processes, excess sugar has a number of other significant consequences.

Nancy Appleton, PhD, author of the book Lick the Sugar Habit[5], contributed an extensive list of the many ways sugar can ruin your health from a vast number of medical journals and other scientific publications.

  1. Sugar can suppress your immune system and impair your defenses against infectious disease.[6] [7]
  2. Sugar upsets the mineral relationships in your body: causes chromium and copper deficiencies and interferes with absorption of calcium and magnesium.[8] [9] [10] [11]
  3. Sugar can cause a rapid rise of adrenaline, hyperactivity, anxiety, difficulty concentrating, and crankiness in children.[12] [13]
  4. Sugar can produce a significant rise in total cholesterol, triglycerides and bad cholesterol and a decrease in good cholesterol.[14][15] [16] [17]
  5. Sugar causes a loss of tissue elasticity and function.[18]
  6. Sugar feeds cancer cells and has been connected with the development of cancer of the breast, ovaries, prostate, rectum, pancreas, biliary tract, lung, gallbladder and stomach.[19] [20] [21] [22] [23] [24] [25]
  7. Sugar can increase fasting levels of glucose and can cause reactive hypoglycemia.[26] [27]
  8. Sugar can weaken eyesight.[28] 1
  9. Sugar can cause many problems with the gastrointestinal tract including: an acidic digestive tract, indigestion, malabsorption in patients with functional bowel disease, increased risk of Crohn’s disease, and ulcerative colitis.[29] [30] [31] [32] [33]
  10. Sugar can cause premature aging.[34] In fact, the single most important factor that accelerates aging is insulin, which is triggered by sugar. 1
  11. Sugar can lead to alcoholism.[35]
  12. Sugar can cause your saliva to become acidic, tooth decay, and periodontal disease.[36] [37] [38]
  13. Sugar contributes to obesity. [39] 1
  14. Sugar can cause autoimmune diseases such as: arthritis, asthma, and multiple sclerosis.[40] [41] [42]
  15. Sugar greatly assists the uncontrolled growth of Candida Albicans (yeast infections) [43]
  16. Sugar can cause gallstones.[44]
  17. Sugar can cause appendicitis.[45]
  18. Sugar can cause hemorrhoids.[46]
  19. Sugar can cause varicose veins.[47]
  20. Sugar can elevate glucose and insulin responses in oral contraceptive users.[48]
  21. Sugar can contribute to osteoporosis.[49]
  22. Sugar can cause a decrease in your insulin sensitivity thereby causing an abnormally high insulin levels and eventually diabetes.[50] [51] [52]
  23. Sugar can lower your Vitamin E levels.[53]
  24. Sugar can increase your systolic blood pressure.[54]
  25. Sugar can cause drowsiness and decreased activity in children.[55]
  26. High sugar intake increases advanced glycation end products (AGEs),which are sugar molecules that attach to and damage proteins in your body. AGEs speed up the aging of cells, which may contribute to a variety of chronic and fatal diseases. [56] 1
  27. Sugar can interfere with your absorption of protein.[57]
  28. Sugar causes food allergies.[58]
  29. Sugar can cause toxemia during pregnancy.[59]
  30. Sugar can contribute to eczema in children.[60]
  31. Sugar can cause atherosclerosis and cardiovascular disease.[61] [62]
  32. Sugar can impair the structure of your DNA.[63]
  33. Sugar can change the structure of protein and cause a permanent alteration of the way the proteins act in your body.[64] [65]
  34. Sugar can make your skin age by changing the structure of collagen.[66]
  35. Sugar can cause cataracts and nearsightedness.[67] [68]
  36. Sugar can cause emphysema.[69]
  37. High sugar intake can impair the physiological homeostasis of many systems in your body.[70]
  38. Sugar lowers the ability of enzymes to function.[71]
  39. Sugar intake is higher in people with Parkinson’s disease.[72]
  40. Sugar can increase the size of your liver by making your liver cells divide, and it can increase the amount of fat in your liver, leading to fatty liver disease.[73] [74]
  41. Sugar can increase kidney size and produce pathological changes in the kidney such as the formation of kidney stones.[75] [76]Fructose is helping to drive up rates of kidney disease. 1
  42. Sugar can damage your pancreas.[77]
  43. Sugar can increase your body’s fluid retention.[78]
  44. Sugar is enemy #1 of your bowel movement.[79]
  45. Sugar can compromise the lining of your capillaries.[80]
  46. Sugar can make your tendons more brittle.[81]
  47. Sugar can cause headaches, including migraines.[82]
  48. Sugar can reduce the learning capacity, adversely affect your children’s grades and cause learning disorders.[83] [84]
  49. Sugar can cause an increase in delta, alpha, and theta brain waves, which can alter your ability to think clearly.[85]
  50. Sugar can cause depression.[86]
  51. Sugar can increase your risk of gout.[87]
  52. Sugar can increase your risk of Alzheimer’s disease.[88] MRI studies show that adults 60 and older who have high uric acid are four to five times more likely to have vascular dementia, the second most common form of dementia after Alzheimer’s.1
  53. Sugar can cause hormonal imbalances such as: increasing estrogen in men, exacerbating PMS, and decreasing growth hormone.[89] [90] [91] [92]
  54. Sugar can lead to dizziness.[93]
  55. Diets high in sugar will increase free radicals and oxidative stress.[94]
  56. A high sucrose diet of subjects with peripheral vascular disease significantly increases platelet adhesion.[95]
  57. High sugar consumption by pregnant adolescents can lead to a substantial decrease in gestation duration and is associated with a twofold-increased risk for delivering a small-for-gestational-age (SGA) infant.[96] [97]
  58. Sugar is an addictive substance.[98]
  59. Sugar can be intoxicating, similar to alcohol.[99]
  60. Sugar given to premature babies can affect the amount of carbon dioxide they produce.[100]
  61. Decrease in sugar intake can increase emotional stability.[101]
  62. Your body changes sugar into 2 to 5 times more fat in the bloodstream than it does starch.[102]
  63. The rapid absorption of sugar promotes excessive food intake in obese subjects.[103]
  64. Sugar can worsen the symptoms of children with attention deficit hyperactivity disorder (ADHD).[104]
  65. Sugar adversely affects urinary electrolyte composition.[105]
  66. Sugar can impair the function of your adrenal glands.[106]
  67. Sugar has the potential of inducing abnormal metabolic processes in normal, healthy individuals, thereby promoting chronic degenerative diseases.[107]
  68. Intravenous feedings (IVs) of sugar water can cut off oxygen to your brain.[108]
  69. Sugar increases your risk of polio.[109]
  70. High sugar intake can cause epileptic seizures.[110]
  71. Sugar causes high blood pressure in obese people.[111]
  72. In intensive care units, limiting sugar saves lives.[112]
  73. Sugar may induce cell death.[113]
  74. In juvenile rehabilitation centers, when children were put on low sugar diets, there was a 44 percent drop in antisocial behavior.[114]
  75. Sugar dehydrates newborns.[115]
  76. Sugar can cause gum disease.[116]

It should now be crystal clear just how damaging sugar is. You simply cannot achieve your highest degree of health and vitality if you are consuming a significant amount of it.

Fortunately, your body has an amazing ability to heal itself when given the basic nutrition it needs, and your liver has an incredible ability to regenerate. If you start making changes today, your health WILL begin to improve, returning you to the state of vitality that nature intended.

What Else Does the Science Say about the Health Impact of Fructose?

According to GreenMedInfo.com, scientific studies have linked fructose to about 30 different specific diseases and health problems. Select the hyperlinks provided to review how fructose may:

Raise your blood pressure, and cause nocturnal hypertension Insulin resistance / Type 2 Diabetes Non-alcoholic fatty liver disease(NAFLD)
Raise your uric acid levels, which can result in gout and/ormetabolic syndrome Accelerate the progression of chronic kidney disease Intracranial atherosclerosis(narrowing and hardening of the arteries in your skull)
Exacerbate cardiac abnormalities if you’re deficient in copper Have a genotoxic effect on the colon Promote metastasis in breast cancer patients
Cause tubulointerstitial injury(injury to the tubules and interstitial tissue of your kidney) Promotes obesity and related health problems and diseases Promotes pancreatic cancer growth

References:

  • [1] Johnson RJ and Gower T. (2009) The Sugar Fix: The High-Fructose Fallout That is Making You Sick and Fat, Pocket, 416 pp
  • [2] ”What sweetener should you choose? Sugar? Honey? Agave nectar?“ Fitnessspotlight
  • [3] Stanhope KL, Schwarz JM, Keim NL, Griffen SC, Bremer AA, Graham JL, Hatcher B, Cox CL, Dyachenko A, Zhang W, McGahan JP, Seibert A, Krauss RM, Chiu S, Schaefer EJ, Ai M, Otokozawa S, Nakajima K, Nakano T, Beysen C, Hellerstein MK, Berglund L and Havel PJ. “Consuming fructose-sweetened, not glucose-sweetened, beverages increases visceral adiposity and lipids and decreases insulin sensitivity in overweight/obese humans,” J Clin Invest. 2009; 119(5):1322-1334
  • [4] Park A. “All sugars aren’t the same: Glucose is better, study says,” Time Magazine, April 21, 2009
  • [5] Appleton N. Lick the Sugar Habit (1996) Avery, 2nd Ed. 272 pp.
  • [6] Sanchez, A., et al. Role of Sugars in Human Neutrophilic Phagocytosis, American Journal of Clinical Nutrition. Nov 1973;261:1180_1184. Bernstein, J., al. Depression of Lymphocyte Transformation Following Oral Glucose Ingestion. American Journal of Clinical Nutrition.1997;30:613
  • [7] Ringsdorf, W., Cheraskin, E. and Ramsay R. Sucrose, Neutrophilic Phagocytosis and Resistance to Disease, Dental Survey. 1976;52(12):46_48
  • [8] Couzy, F., et al. “Nutritional Implications of the Interaction Minerals,” Progressive Food and Nutrition Science 17;1933:65-87
  • [9] Kozlovsky, A., et al. Effects of Diets High in Simple Sugars on Urinary Chromium Losses. Metabolism. June 1986;35:515_518
  • [10] Fields, M.., et al. Effect of Copper Deficiency on Metabolism and Mortality in Rats Fed Sucrose or Starch Diets, Journal of Clinical Nutrition. 1983;113:1335_1345
  • [11] Lemann, J. Evidence that Glucose Ingestion Inhibits Net Renal Tubular Reabsorption of Calcium and Magnesium. Journal of Clinical Nutrition. 1976 ;70:236_245
  • [12] Goldman, J., et al. Behavioral Effects of Sucrose on Preschool Children. Journal of Abnormal Child Psychology.1986;14(4):565_577
  • [13] Jones, T. W., et al. Enhanced Adrenomedullary Response and Increased Susceptibility to Neuroglygopenia: Mechanisms Underlying the Adverse Effect of Sugar Ingestion in Children. Journal of Pediatrics. Feb 1995;126:171-7
  • [14] Scanto, S. and Yudkin, J. The Effect of Dietary Sucrose on Blood Lipids, Serum Insulin, Platelet Adhesiveness and Body Weight in Human Volunteers, Postgraduate Medicine Journal. 1969;45:602_607
  • [15] Albrink, M. and Ullrich I. H. Interaction of Dietary Sucrose and Fiber on Serum Lipids in Healthy Young Men Fed High Carbohydrate Diets. American Journal of Clinical Nutrition. 1986;43:419­
  • [16] Reiser, S. Effects of Dietary Sugars on Metabolic Risk Factors Associated with Heart Disease. Nutritional Health. 1985;203_216
  • [17] Lewis, G. F. and Steiner, G. Acute Effects of Insulin in the Control of Vldl Production in Humans. Implications for The insulin-resistant State. Diabetes Care. 1996 Apr;19(4):390-3 R. Pamplona, M. .J., et al. Mechanisms of Glycation in Atherogenesis. Medical Hypotheses. 1990;40:174-181
  • [18] Cerami, A., Vlassara, H., and Brownlee, M. “Glucose and Aging.” Scientific American. May 1987:90. Lee, A. T. and Cerami, A. The Role of Glycation in Aging. Annals of the New York Academy of Science; 663:63-67
  • [19] Takahashi, E., Tohoku University School of Medicine, Wholistic Health Digest. October 1982:41:00
  • [20] Quillin, Patrick, Cancer’s Sweet Tooth, Nutrition Science News. Ap 2000 Rothkopf, M.. Nutrition. July/Aug 1990;6(4)
  • [21] Michaud, D. Dietary Sugar, Glycemic Load, and Pancreatic Cancer Risk in a Prospective Study. J Natl Cancer Inst. Sep 4, 2002 ;94(17):1293-300
  • [22] Moerman, C. J., et al. Dietary Sugar Intake in the Etiology of Biliary Tract Cancer. International Journal of Epidemiology. Ap 1993.2(2):207-214.
  • [23] The Edell Health Letter. Sept 1991;7:1
  • [24] De Stefani, E.”Dietary Sugar and Lung Cancer: a Case control Study in Uruguay.” Nutrition and Cancer. 1998;31(2):132_7
  • [25] Cornee, J., et al. A Case-control Study of Gastric Cancer and Nutritional Factors in Marseille, France. European Journal of Epidemiology 11 (1995):55-65
  • [26] Kelsay, J., et al. Diets High in Glucose or Sucrose and Young Women. American Journal of Clinical Nutrition. 1974;27:926_936. Thomas, B. J., et al. Relation of Habitual Diet to Fasting Plasma Insulin Concentration and the Insulin Response to Oral Glucose, Human Nutrition Clinical Nutrition. 1983; 36C(1):49_51
  • [27] Dufty, William. Sugar Blues. (New York:Warner Books, 1975)
  • [28] Acta Ophthalmologica Scandinavica. Mar 2002;48;25. Taub, H. Ed. Sugar Weakens Eyesight, VM NEWSLETTER;May 1986:06:00
  • [29] Dufty.
  • [30] Yudkin, J. Sweet and Dangerous.(New York:Bantam Books,1974) 129
  • [31] Cornee, J., et al. A Case-control Study of Gastric Cancer and Nutritional Factors in Marseille, France, European Journal of Epidemiology. 1995;11
  • [32] Persson P. G., Ahlbom, A., and Hellers, G. Epidemiology. 1992;3:47-52
  • [33] Jones, T. W., et al. Enhanced Adrenomedullary Response and Increased Susceptibility to Neuroglygopenia: Mechanisms Underlying the Adverse Effect of Sugar Ingestion in Children. Journal of Pediatrics. Feb 1995;126:171-7
  • [34] Lee, A. T.and Cerami A. The Role of Glycation in Aging. Annals of the New York Academy of Science.1992;663:63-70
  • [35] Abrahamson, E. and Peget, A. Body, Mind and Sugar. (New York: Avon, 1977)
  • [36] Glinsmann, W., Irausquin, H., and Youngmee, K. Evaluation of Health Aspects of Sugar Contained in Carbohydrate Sweeteners. F. D. A. Report of Sugars Task Force. 1986:39:00 Makinen K.K.,et al. A Descriptive Report of the Effects of a 16_month Xylitol Chewing_gum Programme Subsequent to a 40_month Sucrose Gum Programme. Caries Research. 1998; 32(2)107_12
  • [37] Glinsmann, W., Irausquin, H., and K. Youngmee. Evaluation of Health Aspects of Sugar Contained in Carbohydrate Sweeteners. F. D. A. Report of Sugars Task Force.1986;39:36_38
  • [38] Appleton, N. New York: Healthy Bones. Avery Penguin Putnam:1989
  • [39] Keen, H., et al. Nutrient Intake, Adiposity, and Diabetes. British Medical Journal. 1989; 1:00 655_658
  • [40] Darlington, L., Ramsey, N. W. and Mansfield, J. R. Placebo Controlled, Blind Study of Dietary Manipulation Therapy in Rheumatoid Arthritis, Lancet. Feb 1986;8475(1):236_238
  • [41] Powers, L. Sensitivity: You React to What You Eat. Los Angeles Times. (Feb. 12, 1985). Cheng, J., et al. Preliminary Clinical Study on the Correlation Between Allergic Rhinitis and Food Factors. Lin Chuang Er Bi Yan Hou Ke Za Zhi Aug 2002;16(8):393-396
  • [42] Erlander, S. The Cause and Cure of Multiple Sclerosis, The Disease to End Disease.” Mar 3, 1979;1(3):59_63
  • [43] Crook, W. J. The Yeast Connection. (TN:Professional Books, 1984)
  • [44] Heaton, K. The Sweet Road to Gallstones. British Medical Journal. Apr 14, 1984; 288:00:00 1103_1104. Misciagna, G., et al. American Journal of Clinical Nutrition. 1999;69:120-126
  • [45] Cleave, T. The Saccharine Disease. (New Canaan, CT: Keats Publishing, 1974)
  • [46] Ibid
  • [47] Cleave, T. and Campbell, G. (Bristol, England:Diabetes, Coronary Thrombosis and the Saccharine Disease: John Wright and Sons, 1960)
  • [48] Behall, K. Influ ence of Estrogen Content of Oral Contraceptives and Consumption of Sucrose on Blood Parameters. Disease Abstracts International. 1982;431437
  • [49] Tjäderhane, L. and Larmas, M. A High Sucrose Diet Decreases the Mechanical Strength of Bones in Growing Rats. Journal of Nutrition. 1998:128:1807_1810
  • [50] Beck, Nielsen H., Pedersen O., and Schwartz S. Effects of Diet on the Cellular Insulin Binding and the Insulin Sensitivity in Young Healthy Subjects. Diabetes. 1978;15:289_296
  • [51] Sucrose Induces Diabetes in Cat. Federal Protocol. 1974;6(97). diabetes
  • [52] Reiser, S., et al. Effects of Sugars on Indices on Glucose Tolerance in Humans. American Journal of Clinical Nutrition. 1986;43:151-159
  • [53] Journal of Clinical Endocrinology and Metabolism. Aug 2000
  • [54] Hodges, R., and Rebello, T. Carbohydrates and Blood Pressure. Annals of Internal Medicine. 1983:98:838_841
  • [55] Behar, D., et al. Sugar Challenge Testing with Children Considered Behaviorally Sugar Reactive. Nutritional Behavior. 1984;1:277_288
  • [56] Furth, A. and Harding, J. Why Sugar Is Bad For You. New Scientist. Sep 23, 1989;44
  • [57] Simmons, J. Is The Sand of Time Sugar? LONGEVITY. June 1990:00:00 49_53
  • [58] Appleton, N. New York: LICK THE SUGAR HABIT. Avery Penguin Putnam:1988. allergies
  • [59] Cleave, T. The Saccharine Disease: (New Canaan Ct: Keats Publishing, Inc., 1974).131
  • [60] Ibid. 132
  • [61] Pamplona, R., et al. Mechanisms of Glycation in Atherogenesis. Medical Hypotheses . 1990:00:00 174_181
  • [62] Vaccaro O., Ruth, K. J. and Stamler J. Relationship of Postload Plasma Glucose to Mortality with 19 yr Follow up. Diabetes Care. Oct 15,1992;10:328_334. Tominaga, M., et al, Impaired Glucose Tolerance Is a Risk Factor for Cardiovascular Disease, but Not Fasting Glucose. Diabetes Care. 1999:2(6):920-924
  • [63] Lee, A. T. and Cerami, A. Modifications of Proteins and Nucleic Acids by Reducing Sugars: Possible Role in Aging. Handbook of the Biology of Aging. (New York: Academic Press, 1990)
  • [64] Monnier, V. M. Nonenzymatic Glycosylation, the Maillard Reaction and the Aging Process. Journal of Gerontology 1990:45(4):105_110
  • [65] Cerami, A., Vlassara, H., and Brownlee, M. Glucose and Aging. Scientific American. May 1987:00:00 90
  • [66] Dyer, D. G., et al. Accumulation of Maillard Reaction Products in Skin Collagen in Diabetes and Aging. Journal of Clinical Investigation. 1993:93(6):421_22
  • [67] Veromann, S.et al.”Dietary Sugar and Salt Represent Real Risk Factors for Cataract Development.” Ophthalmologica. 2003 Jul-Aug;217(4):302-307
  • [68] Goulart, F. S. Are You Sugar Smart? American Fitness. March_April 1991:00:00 34_38. Milwakuee, WI
  • [69] Monnier, V. M. Nonenzymatic Glycosylation, the Maillard Reaction and the Aging Process. Journal of Gerontology. 1990:45(4):105_110
  • [70] Ceriello, A. Oxidative Stress and Glycemic Regulation. Metabolism. Feb 2000;49(2 Suppl 1):27­29
  • [71] Appleton, Nancy. New York; Lick the Sugar Habit. Avery Penguin Putnam, 1988 enzymes
  • [72] Hellenbrand, W. Diet and Parkinson’s Disease. A Possible Role for the Past Intake of Specific Nutrients. Results from a Self-administered Food-frequency Questionnaire in a Case-control Study. Neurology. Sep 1996;47(3):644-650
  • [73] Goulart, F. S. Are You Sugar Smart? American Fitness. March_April 1991:00:00 34_38
  • [74] Ibid.
  • [75] Yudkin, J., Kang, S. and Bruckdorfer, K. Effects of High Dietary Sugar. British Journal of Medicine. Nov 22, 1980;1396
  • [76] Blacklock, N. J., Sucrose and Idiopathic Renal Stone. Nutrition and Health. 1987;5(1-2):9­Curhan, G., et al. Beverage Use and Risk for Kidney Stones in Women. Annals of Internal Medicine. 1998:28:534-340
  • [77] Goulart, F. S. Are You Sugar Smart? American Fitness. March_April 1991:00:00 34_38. Milwakuee, WI
  • [78] Ibid. fluid retention
  • [79] Ibid. bowel movement
  • [80] Ibid. compromise the lining of the capillaries
  • [81] Nash, J. Health Contenders. Essence. Jan 1992; 23:00 79_81
  • [82] Grand, E. Food Allergies and Migraine.Lancet. 1979:1:955_959
  • [83] Schauss, A. Diet, Crime and Delinquency. (Berkley Ca; Parker House, 1981)
  • [84] Molteni, R, et al. A High-fat, Refined Sugar Diet Reduces Hippocampal Brain-derived Neurotrophic Factor, Neuronal Plasticity, and Learning. NeuroScience. 2002;112(4):803-814
  • [85] Christensen, L. The Role of Caffeine and Sugar in Depression. Nutrition Report. Mar 1991;9(3):17-24
  • [86] Ibid,44
  • [87] Yudkin, J. Sweet and Dangerous.(New York:Bantam Books,1974) 129
  • [88] Frey, J. Is There Sugar in the Alzheimer’s Disease? Annales De Biologie Clinique. 2001; 59 (3):253-257
  • [89] Yudkin, J. Metabolic Changes Induced by Sugar in Relation to Coronary Heart Disease and Diabetes. Nutrition and Health. 1987;5(1-2):5-8
  • [90] Yudkin, J and Eisa, O. Dietary Sucrose and Oestradiol Concentration in Young Men. Annals of Nutrition and Metabolism. 1988:32(2):53-55
  • [91] The Edell Health Letter. Sept 1991;7:1
  • [92] Gardner, L. and Reiser, S. Effects of Dietary Carbohydrate on Fasting Levels of Human Growth Hormone and Cortisol. Proceedings of the Society for Experimental Biology and Medicine. 1982;169:36_40
  • [93] Journal of Advanced Medicine. 1994;7(1):51-58
  • [94] Ceriello, A. Oxidative Stress and Glycemic Regulation. Metabolism. Feb 2000;49(2 Suppl 1):27­29
  • [95] Postgraduate Medicine.Sept 1969:45:602-07
  • [96] Lenders, C. M. Gestational Age and Infant Size at Birth Are Associated with Dietary Intake among Pregnant Adolescents. Journal of Nutrition. Jun 1997;1113-1117
  • [97] Ibid.
  • [98] Sugar, White Flour Withdrawal Produces Chemical Response. The Addiction Letter. Jul 1992:04:00 Colantuoni, C., et al. Evidence That Intermittent, Excessive Sugar Intake Causes Endogenous Opioid Dependence. Obes Res. Jun 2002 ;10(6):478-488. Annual Meeting of the American Psychological Society, Toronto, June 17, 2001 www.mercola.com/2001/jun/30/sugar.htm
  • [99] Ibid.
  • [100] Sunehag, A. L., et al. Gluconeogenesis in Very Low Birth Weight Infants Receiving Total Parenteral Nutrition Diabetes. 1999 ;48 7991_800
  • [101] Christensen L., et al. Impact of A Dietary Change on Emotional Distress. Journal of Abnormal Psychology.1985;94(4):565_79
  • [102] Nutrition Health Review. Fall 85 changes sugar into fat faster than fat
  • [103] Ludwig, D. S., et al. High Glycemic Index Foods, Overeating and Obesity. Pediatrics. March 1999;103(3):26-32
  • [104] Pediatrics Research. 1995;38(4):539-542. Berdonces, J. L. Attention Deficit and Infantile Hyperactivity. Rev Enferm. Jan 2001;4(1)11-4
  • [105] Blacklock, N. J. Sucrose and Idiopathic Renal Stone. Nutrition Health. 1987;5(1 & 2):9­
  • [106] Lechin, F., et al. Effects of an Oral Glucose Load on Plasma Neurotransmitters in Humans. Neurophychobiology. 1992;26(1-2):4-11
  • [107] Fields, M. Journal of the American College of Nutrition. Aug 1998;17(4):317_321
  • [108] Arieff, A. I. Veterans Administration Medical Center in San Francisco. San Jose Mercury; June 12/86. IVs of sugar water can cut off oxygen to the brain
  • [109] Sandler, Benjamin P. Diet Prevents Polio. Milwakuee, WI,:The Lee Foundation for for Nutritional Research, 1951
  • [110] Murphy, Patricia. The Role of Sugar in Epileptic Seizures. Townsend Letter for Doctors and Patients. May, 2001 Murphy Is Editor of Epilepsy Wellness Newsletter, 1462 West 5th Ave., Eugene, Oregon 97402
  • [111] Stern, N. & Tuck, M. Pathogenesis of Hypertension in Diabetes Mellitus. Diabetes Mellitus, a Fundamental and Clinical Test. 2nd Edition, (PhiladelphiA; A:Lippincott Williams & Wilkins, 2000)943-957
  • [112] Christansen, D. Critical Care: Sugar Limit Saves Lives. Science News. June 30, 2001; 159:404
  • [113] Donnini, D. et al. Glucose May Induce Cell Death through a Free Radical-mediated Mechanism.Biochem Biohhys Res Commun. Feb 15, 1996:219(2):412-417
  • [114] Schoenthaler, S. The Los Angeles Probation Department Diet-Behavior Program: Am Empirical Analysis of Six Institutional Settings. Int J Biosocial Res 5(2):88-89
  • [115] Gluconeogenesis in Very Low Birth Weight Infants Receiving Total Parenteral Nutrition. Diabetes. 1999 Apr;48(4):791-800
  • [116] Glinsmann, W., et al. Evaluation of Health Aspects of Sugar Contained in Carbohydrate Sweeteners.” FDA Report of Sugars Task Force -1986 39 123Yudkin, J. and Eisa, O. Dietary Sucrose and Oestradiol Concentration in Young Men. Annals of Nutrition and Metabolism. 1988;32(2):53-5
healthy eating is the key hope you got out of this what we saw in it and we can all get back into shape.  Check out our other eating right sections for more valuable tips on diet, nutrition, and eating.